Journal of neuroinflammation

[1] Systemic microbial TLR2 agonists induce neurodegeneration in Alzheimer's disease mice.

[2] TLR2 Expression on Select Lymphocyte Subsets as a New Marker in Glomerulonephritis

[3] tlr2 signaling is required for the innate, but not adaptive response to lvs clpb

[4] phosphatidyl-inositol-3 kinase inhibitors regulate peptidoglycan-induced myeloid leukocyte recruitment, inflammation, and neurotoxicity in mouse brain

[5] association between arg753gln and arg677trp polymorphisms of tlr2 gene with active pulmonary tuberculosis in an indonesian population

[6] toll-like receptor 2 mediates in vivo pro- and anti-inflammatory effects of mycobacterium tuberculosis and modulates autoimmune encephalomyelitis

[7] tlr2−/− mice display decreased severity of giardiasis via enhanced proinflammatory cytokines production dependent on akt signal pathway

[8] Functional and genetic evidence that the Mal/TIRAP allele variant 180L has been selected by providing protection against septic shock

[9] Extracellular vesicles from CLEC2-activated platelets enhance dengue virus-induced lethality via CLEC5A/TLR2

[10] Systemic microbial TLR2 agonists induce neurodegeneration in Alzheimer's disease mice.

[11] Pathogen-Associated Molecular Pattern-Induced TLR2 and TLR4 Activation Increases Keratinocyte Production of Inflammatory Mediators and is Inhibited by Phosphatidylglycerol.

[12] TLR2 Expression on Select Lymphocyte Subsets as a New Marker in Glomerulonephritis

[13] 17β-Estradiol Protects the Retinal Nerve Cells Suppressing TLR2 Mediated Immune-Inflammation and Apoptosis from Oxidative Stress Insult Independent of PI3K

[14] targeting of immune cells by dual tlr2/7 ligands suppresses features of allergic th2 immune responses in mice

[15] cholesterol oxidase binds tlr2 and modulates functional responses of human macrophages

[16] tlr2 expression in peripheral cd4+ t cells promotes th17 response and is associated with disease aggravation of hepatitis b virus-related acute-on-chronic liver failure

[17] tlr2 signaling is required for the innate, but not adaptive response to lvs clpb

[18] cigarette smoke amplifies inflammatory response and atherosclerosis progression through activation of the h1r-tlr2/4-cox2 axis

[19] association between arg753gln and arg677trp polymorphisms of tlr2 gene with active pulmonary tuberculosis in an indonesian population

[20] decreased secondary lesion growth and attenuated immune response after traumatic brain injury in tlr2/4−/− mice

[21] tlr2−/− mice display decreased severity of giardiasis via enhanced proinflammatory cytokines production dependent on akt signal pathway