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Keyword Connections - transgenic mice Keywords

Connection Type	Connection
Journals	Journal of Alzheimer's disease : JAD advances in medical sciences
Research Groups	No Research Group Connected
Bibliographies	[1] Specific overexpression of 15-lipoxygenase in endothelial cells promotes cancer cell death in an in vivo Lewis lung carcinoma mouse model. [2] A technique for simultaneous measurement of Ca2+, FRET fluorescence and force in intact mouse small arteries [3] conditional cripto overexpression in satellite cells promotes myogenic commitment and enhances early regeneration [4] mapping transgene insertion sites reveals complex interactions between mouse transgenes and neighboring endogenous genes [5] hippocampal cholinergic interneurons visualized with the choline acetyltransferase promoter: anatomical distribution, intrinsic membrane properties, neurochemical characteristics, and capacity for cholinergic modulation [6] synaptic function is modulated by lrrk2 and glutamate release is increased in cortical neurons of g2019s lrrk2 knock-in mice [7] modulation of specific sensory cortical areas by segregated basal forebrain cholinergic neurons demonstrated by neuronal tracing and optogenetic stimulation in mice [8] proteasome inhibition slightly improves cardiac function in mice with hypertrophic cardiomyopathy [9] Fecal microbiota transplantation alleviated Alzheimer’s disease-like pathogenesis in APP/PS1 transgenic mice [10] Genotype-induced changes in biophysical properties of frontal cortex lipid raft from APP/PS1 transgenic mice [11] A transplantable tumor model allowing investigation of NY-BR-1-specific T cell responses in HLA-DRB1*0401 transgenic mice. [12] Role of Esrrg in the fibrate-mediated regulation of lipid metabolism genes in human ApoA-I transgenic mice [13] Phosphorylated tau in neuritic plaques of APP sw /Tau vlw transgenic mice and Alzheimer disease [14] Environmental neurotoxic challenge of conditional alpha-synuclein transgenic mice predicts a dopaminergic olfactory-striatal interplay in early PD [15] tau depletion in app transgenic mice attenuates task-related hyperactivation of the hippocampus and differentially influences locomotor activity and spatial memory [16] mangifera indica l. extract (vimang) improves the aversive memory in spinocerebellar ataxia type 2 transgenic mice. [17] incidental abnormalities in oral mucosal carcinogenesis of v-h-ras transgenic mice of v-h-ras transgenic mice [18] stromal activation associated with development of prostate cancer in prostate-targeted fibroblast growth factor 8b transgenic mice [19] increased astrocyte expression of il-6 or ccl2 in transgenic mice alters levels of hippocampal and cerebellar proteins [20] a soluble form of human nectin-2 impairs exocrine secretion of pancreas and formation of zymogen granules in transgenic mice [21] comparison of expression profiles of metastatic versus primary mammary tumors in mmtv-wnt-1 and mmtv-neu transgenic mice [22] next-generation transgenic mice for optogenetic analysis of neural circuits [23] transplantation of human menstrual blood-derived mesenchymal stem cells alleviates alzheimer’s disease-like pathology in app/ps1 transgenic mice [24] immunohistochemical evidence from app-transgenic mice for glutaminyl cyclase as drug target to diminish pe-abeta formation [25] vaccine development to treat alzheimer’s disease neuropathology in app/ps1 transgenic mice [26] Age-Related Changes in Body Composition of Bovine Growth Hormone Transgenic Mice [27] Growth Hormone Receptor Antagonist Transgenic Mice Are Protected From Hyperinsulinemia and Glucose Intolerance Despite Obesity When Placed on a HF Diet [28] Elevated Systolic Blood Pressure in Male GH Transgenic Mice Is Age Dependent [29] Male Bovine GH Transgenic Mice Have Decreased Adiposity With an Adipose Depot-Specific Increase in Immune Cell Populations [30] Insulin resistance and diabetes mellitus in transgenic mice expressing nuclear SREBP-1c in adipose tissue: model for congenital generalized lipodystrophy [31] Isoform 1c of sterol regulatory element binding protein is less active than isoform 1a in livers of transgenic mice and in cultured cells [32] The c-myc oncogene driven by immunoglobulin enhancers induces lymphoid malignancy in transgenic mice [33] Isoform 1c of sterol regulatory element binding protein is less active than isoform 1a in livers of transgenic mice and in cultured cells. [34] Overexpression of bone morphogenetic protein-6 (BMP-6) in the epidermis of transgenic mice: inhibition or stimulation of proliferation depending on the pattern of transgene expression and formation of psoriatic lesions. [35] Spreading of TDP-43 pathology via pyramidal tract induces ALS-like phenotypes in TDP-43 transgenic mice [36] Electrophysiology of Arcuate Neurokinin B Neurons in Female Tac2-EGFP Transgenic Mice [37] Vanin-1 Inactivation Antagonizes the Development of Adrenocortical Neoplasia in Sf-1 Transgenic Mice [38] Isoform 1c of sterol regulatory element binding protein is less active than isoform 1a in livers of transgenic mice and in cultured cells. [39] Transgenic mice as a model to study the role of TGF-beta-related molecules in hair follicles. [40] Insulin resistance and diabetes mellitus in transgenic mice expressing nuclear SREBP-1c in adipose tissue: model for congenital generalized lipodystrophy [41] Cell-type-specific DNA methylation analysis of the frontal cortices of mutant Polg1 transgenic mice with neuronal accumulation of deleted mitochondrial DNA.