International journal of molecular sciences

genome research

faraday discussions

[1] Unravelling the Metabolic and Hormonal Machinery During Key Steps of Somatic Embryogenesis: A Case Study in Coffee.

[2] Temperature dependence of the vibrational spectrum of porphycene: a qualitative failure of classical-nuclei molecular dynamics.

[3] Transcriptional alterations in glioma result primarily from DNA methylation-independent mechanisms.

[4] direct reprogramming of adult somatic cells towards adventitious root formation in forest tree species: the effect of the juvenile-adult transition

[5] ezrin: a regulator of actin microfilaments in cell junctions of the rat testis

[6] reprogramming cell fate: a changing story

[7] postembryonic organogenesis and plant regeneration from tissues:two sides of the same coin?

[8] Modeling reveals bistability and low-pass filtering in the network module determining blood stem cell fate.

[9] Computational Tools for Quantifying Concordance in Single-Cell Fate.

[10] Single-Cell Landscape of Transcriptional Heterogeneity and Cell Fate Decisions during Mouse Early Gastrulation

[11] Subcellular Control over Focal Adhesion Anisotropy, Independent of Cell Morphology, Dictates Stem Cell Fate.

[12] p53 pulse modulation differentially regulates target gene promoters to regulate cell fate decisions.

[13] dynamic notch signaling specifies each cell fate in drosophila spermathecal lineage

[14] micrornas: key triggers of neuronal cell fate

[15] reprogramming cell fate: a changing story

[16] the bile acid nuclear receptor fxrα is a critical regulator of mouse germ cell fate

[17] pigment epithelium-derived factor (pedf) is a determinant of stem cell fate: lessons from an ultra-rare disease

[18] regulation of cardiac cell fate by micrornas: implications for heart regeneration

[19] cell fate and differentiation of bone marrow mesenchymal stem cells

[20] Lymphoid enhancer factor 1 directs hair follicle patterning and epithelial cell fate.

[21] Genetic rescue of Muenke syndrome model hearing loss reveals prolonged FGF-dependent plasticity in cochlear supporting cell fates