Environmental pollution (Barking, Essex : 1987)

Ecotoxicology and environmental safety

International journal of molecular sciences

Chemosphere

[1] Maternal Exposure to Bisphenol A Combined with High-Fat Diet-Induced Programmed Hypertension in Adult Male Rat Offspring: Effects of Resveratrol.

[2] Inflammatory marker and aryl hydrocarbon receptor-dependent responses in human macrophages exposed to emissions from biodiesel fuels.

[3] quaking deficiency amplifies inflammation in experimental endotoxemia via the aryl hydrocarbon receptor/signal transducer and activator of transcription 1–nf-κb pathway

[4] the aryl hydrocarbon receptor and tumor immunity

[5] role of the aryl hydrocarbon receptor in colon neoplasia

[6] indoxyl sulfate promotes macrophage il-1β production by activating aryl hydrocarbon receptor/nf-κ/mapk cascades, but the nlrp3 inflammasome was not activated

[7] Benzo(a)pyrene induces MUC5AC expression through the AhR/mitochondrial ROS/ERK pathway in airway epithelial cells.

[8] A mixture of persistent organic pollutants relevant for human exposure inhibits the transactivation activity of the aryl hydrocarbon receptor in vitro.

[9] Inflammatory marker and aryl hydrocarbon receptor-dependent responses in human macrophages exposed to emissions from biodiesel fuels.

[10] quaking deficiency amplifies inflammation in experimental endotoxemia via the aryl hydrocarbon receptor/signal transducer and activator of transcription 1–nf-κb pathway

[11] aryl hydrocarbon receptor (ahr)-regulated transcriptomic changes in rats sensitive or resistant to major dioxin toxicities

[12] species traits predict the aryl hydrocarbon receptor 1 (ahr1) subtypes responsible for dioxin sensitivity in birds

[13] the aryl hydrocarbon receptor and tumor immunity

[14] association of aryl hydrocarbon receptor-related gene variants with the severity of autism spectrum disorders

[15] aryl hydrocarbon receptor deficiency in an exon 3 deletion mouse model promotes hematopoietic stem cell proliferation and impacts endosteal niche cells

[16] estrogen receptor α and aryl hydrocarbon receptor cross-talk in a transfected hepatoma cell line (hepg2) exposed to 2,3,7,8-tetrachlorodibenzo-p-dioxin

[17] role of the aryl hydrocarbon receptor in colon neoplasia

[18] the role of aryl hydrocarbon receptor and crosstalk with estrogen receptor in response of breast cancer cells to the novel antitumor agents benzothiazoles and aminoflavone

[19] craniofacial form is altered by chronic adult exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (tcdd) in han/wistar and long–evans rats with different aryl hydrocarbon receptor (ahr) structures

[20] indoxyl sulfate promotes macrophage il-1β production by activating aryl hydrocarbon receptor/nf-κ/mapk cascades, but the nlrp3 inflammasome was not activated