Androgen excess fetal programming of female reproduction: a developmental aetiology for polycystic ovary syndrome?

البرمجة الجنينية للوظيفة الإنجابية الأنثوية بفعل فرط الأندروجين: سببية نمائية لمتلازمة المبيض متعدد الكيسات؟

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ID: 118859
1970
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Abstrak
The aetiology of polycystic ovary syndrome (PCOS) remains unknown. This familial syndrome is prevalent among reproductive-aged women and its inheritance indicates a dominant regulatory gene with incomplete penetrance. However, promising candidate genes have proven unreliable as markers for the PCOS …
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Penulis Abbott DH;Barnett DK;Bruns CM;Dumesic DA;;
Jurnal human reproduction update
Tahun 1970
DOI
DOI tidak ditemukan
URL
http://www.ncbi.nlm.nih.gov/pubmed/15941725
Kata Kunci
National Center for Biotechnology Information NCBI NLM MEDLINE review animals humans pubmed abstract nih national institutes of health national library of medicine research support u.s. gov't female N.I.H. Extramural polycystic ovary syndrome / physiopathology* P.H.S. Hyperandrogenism / complications* d a dumesic d h abbott polycystic ovary syndrome / etiology* genitalia androgens / physiology* pmid:15941725 doi:10.1093/humupd/dmi013 d k barnett female / abnormalities* female / physiopathology hyperandrogenism / physiopathology*

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