Fibroblast growth factor 21 promotes re-epithelialization through the ERK pathways during wound healing

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ID: 333517
2026
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Abstract
Abstract We examined the effects of fibroblast growth factor (FGF)21 on re-epithelialization during wound healing. Wound closure and re-epithelialization were significantly delayed in FGF21 KO mice. For the in vitro assay, the FGF21-induced keratinocyte migration was reversed by extracellular signal-regulated kinase (ERK) inhibitors. These results suggest that FGF21 promotes re-epithelialization via the ERK pathways.
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openalex_W7223227084 Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Takumi Yamane, Ryosuke Konno, Manami Kimura, Okubo Mizuki, Yoshiaki Nakayama, Morichika Konishi, Yuichi Oishi
Journal bioscience biotechnology and biochemistry
Year 2026
DOI
10.1093/bbb/zbag157
URL
https://doi.org/10.1093/bbb/zbag157
Keywords Keywords not found

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