Bullous pemphigoid needs two axes, not one: refining the actionable inflammatory endotype

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ID: 320848
2026
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Abstract
Commenting on Cao et al., we argue that their inflammatory phenotype of bullous pemphigoid is better characterised as IL-13/alarmin-driven than eosinophil-defined—favouring IL-4Rα blockade over eosinophil-directed therapy—and that the reciprocal phenotype should not be assumed low-risk. We therefore propose a complementary autoantibody/complement axis alongside the type 2 axis, with prospective multicentre calibration linking both to differential treatment response as the necessary next step.
Reference Key
openalex_W7168158415 Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Menglu Zhang, Biao Song
Journal the british journal of dermatology
Year 2026
DOI
10.1093/bjd/ljag278
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