Targeting Porphyromonas gingivalis virulence and viability with epimedokoreanin B prevents alveolar bone destruction in vivo

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ID: 315076
2026
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Abstract
Abstract Periodontal disease is a major oral infectious condition that leads to the degradation of alveolar bone and subsequent tooth loss. Porphyromonas gingivalis, a keystone pathogen in periodontitis, plays a critical role in disease initiation and progression. In a previous study, we demonstrated the anti-P. gingivalis activity of epimedokoreanin B, a prenylated flavonoid derived from Epimedium species. However, its in vivo efficacy against periodontitis remains unclear. Oral administration of epimedokoreanin B attenuated P. gingivalis-induced alveolar bone resorption in murine periodontitis models and suppressed infection-induced antibody production Furthermore, epimedokoreanin B compromised bacterial membrane integrity and virulent gene expressions. In this preclinical study, in vitro and in vivo analyses indicate that epimedokoreanin B suppressed both bacterial viability and virulence, thereby attenuating alveolar bone loss caused by periodontitis. These findings suggest that epimedokoreanin B is a promising candidate for the development of novel therapeutics targeting periodontal pathogens.
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openalex_W7162524961 Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Toru Kariu, Keisuke Nakashima, Tsuyoshi Ikeda
Journal bioscience biotechnology and biochemistry
Year 2026
DOI
10.1093/bbb/zbag075
URL
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