Mechanisms underlying the development of androgen-independent prostate cancer

Clicks: 292
ID: 274532
2006
Free PDF
Article Quality & Performance Metrics
Overall Quality
Not rated
Combines reader engagement with the AI quality analysis. This article has not been analysed, so there is no overall score — reader engagement is measured and shown alongside.
AI Quality Assessment
Not analyzed
Readership in this journal
Popular

Ranked #10 of 11 articles by views in clinical cancer research : an official journal of the american association for cancer research

Most read Least read

Bar heights use a square-root scale.

Mint this article as an NFT
Not yet minted

Create a permanent, verifiable on-chain record of this article on the Scimatic Network. The NFT is held in your Journament account, and you can withdraw it to your own wallet at any time.

5 SUSD one-off · no wallet required
Sign in to mint
Abstract
Mechanisms underlying the development of androgen-independent prostate cancer
Reference Key
kj2006clinicalmechanisms Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Pienta KJ;Bradley D;;
Journal clinical cancer research : an official journal of the american association for cancer research
Year 2006
DOI
DOI not found
URL
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Abstract&list_uids=16551847
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE review humans pubmed abstract nih national institutes of health national library of medicine models research support non-u.s. gov't male N.I.H. Extramural Receptors biological signal transduction / physiology* androgens / metabolism androgens / physiology* prostatic neoplasms / metabolism prostatic neoplasms / therapy prostatic neoplasms / physiopathology* pmid:16551847 doi:10.1158/1078-0432.ccr-06-0067 kenneth j pienta deborah bradley androgen / physiology

Citations

No citations found. To add a citation, contact the admin at info@scimatic.org

Comments

No comments yet. Be the first to comment on this article.