alteration of host cell ubiquitination machinery by pathogenic bacteria
Clicks: 294
ID: 244038
2015
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Abstract
Response of immune and non-immune cells to pathogens infections is a very dynamic process. It involves the activation/modulation of many pathways leading to actin remodeling, membrane engulfing, phagocytosis, vesicle trafficking, phagolysosome formation, aiming at the destruction of the intruder. These sophisticated and rapid mechanisms rely on post-translational modifications (PTMs) of key host cells' factors, and bacteria have developed various strategies to manipulate them to favor their survival. Among these important PTMs, ubiquitination has emerged as a major mediator/modulator/regulator of host cells response to infections that pathogens have also learned to use for their own benefit. In this mini-review, we summarize our current knowledge about the normal functions of ubiquitination during host cell infection, and we detail its hijacking by model pathogens to escape clearance and to proliferate.
| Reference Key |
ealomairi2015frontiersalteration
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|---|---|
| Authors | ;Jaafar eAlomairi;Jaafar eAlomairi;Thomas eBonacci;Eric eGhigo;Soubeyran ePhilippe |
| Journal | electronic physician |
| Year | 2015 |
| DOI |
10.3389/fcimb.2015.00017
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| URL | |
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