oxidative stress in cardiovascular diseases and obesity: role of p66shc and protein kinase c

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ID: 242695
2013
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Abstract
Reactive oxygen species (ROS) are a byproduct of the normal metabolism of oxygen and have important roles in cell signalling and homeostasis. An imbalance between ROS production and the cellular antioxidant defence system leads to oxidative stress. Environmental factors and genetic interactions play key roles in oxidative stress mediated pathologies. In this paper, we focus on cardiovascular diseases and obesity, disorders strongly related to each other; in which oxidative stress plays a fundamental role. We provide evidence of the key role played by p66Shc protein and protein kinase C (PKC) in these pathologies by their intracellular regulation of redox balance and oxidative stress levels. Additionally, we discuss possible therapeutic strategies aimed at attenuating the oxidative damage in these diseases.
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marchi2013oxidativeoxidative Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors ;Elena De Marchi;Federica Baldassari;Angela Bononi;Mariusz R. Wieckowski;Paolo Pinton
Journal journal of aoac international
Year 2013
DOI
10.1155/2013/564961
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