the role of infection and inflammation in stillbirths: parallels with sids?

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ID: 167695
2015
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Abstract
It has been suggested that stillbirths are part of the spectrum of infant deaths than includes Sudden Infant Death syndrome (SIDS). This paper examines the hypothesis that risk factors associated with stillbirths might contribution to dysregulation of inflammatory responses to infections that could trigger the physiological events leading to fetal loss. These include genetic factors (ethnic group, sex), environmental (infection, cigarette smoke, obesity) and developmental (testosterone levels) factors. Interactions between the genetic, environmental and developmental risk factors are also considered, e.g., the excess of male stillborn infants in relation to the effects of testosterone levels during development on pro-inflammatory responses. In contrast to SIDS, inflammatory responses of both mother and fetus need to be considered. Approaches for examining the hypothesis are proposed.
Reference Key
eblackwell2015frontiersthe Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors ;Caroline eBlackwell;Caroline eBlackwell
Journal sudebno-meditsinskaia ekspertiza
Year 2015
DOI
10.3389/fimmu.2015.00248
URL
http://journal.frontiersin.org/Journal/10.3389/fimmu.2015.00248/full
Keywords
inflammation infection obesity cigarette smoke stillbirth allergy ethnicityimmunologic diseases

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