isolated deep infarcts: which size indicates single penetrating artery disease?

Clicks: 199
ID: 152224
2007
Free PDF
Article Quality & Performance Metrics
Overall Quality
Not rated
Combines reader engagement with the AI quality analysis. This article has not been analysed, so there is no overall score — reader engagement is measured and shown alongside.
AI Quality Assessment
Not analyzed
Readership in this journal
Star

Ranked #31 of 49 articles by views in primates; journal of primatology

Most read Least read

Bar heights use a square-root scale.

Mint this article as an NFT
Not yet minted

Create a permanent, verifiable on-chain record of this article on the Scimatic Network. The NFT is held in your Journament account, and you can withdraw it to your own wallet at any time.

5 SUSD one-off · no wallet required
Sign in to mint
Abstract
OBJECTIVE: The relationship between infarction size determined in the acute stage and the probability of the presence of large artery stenosis or cardiac source of emboli in patients with isolated deep infarcts was evaluated. METHODS: Maximum transverse diameters of the infarcts seen in 59 consecutive patients’ T2 weighted MRI scans were prospectively recorded. Receiver operating characteristic curves were generated to analyze the optimal size that discriminate IDI with and without underlying large artery stenosis or cardiac source of emboli (LAS/CSE). RESULTS: Twenty-six IDI were in the medial cerebral artery (MCA) territory, 24 in pons and 9 in thalamus. Optimal infarct diameter for MCA IDIs was 25 mm. One of the 10 cases with small (2-14 mm) and half of the 14 cases (50%) with large (17-25 mm) pontine infarcts had LAS/CSE, but only 2 patients, one in each group had severe (≥70%) basilar artery stenosis. None had major CSE. None of the 9 cases with thalamic infarcts (8-20 mm) had LAS/CSE. CONCLUSION: : IDIs in different anatomical locations should be assessed separately. Middle cerebral artery territory IDIs with a maximum diameter of less than 25 mm are rarely associated with LAS/CSE and possibly develop due to occlusion of single lenticulostriate artery. Large unilateral pontine infarcts are usually not associated with severe basilar artery stenosis or major CSE and are probably caused by basilar artery atheromatous branch occlusion
Reference Key
yeilot2007trkisolated Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors ;Nilüfer Yeşilot;Yakup Krespi;Erdem Tüzün;Oğuzhan Çoban;Rezzan Tuncay;Sara Bahar
Journal primates; journal of primatology
Year 2007
DOI
DOI not found
URL
https://www.journalagent.com/tjn/pdfs/TJN_13_3_189_200.pdf
Keywords
etiology diseases of the nervous system atherosclerotic branch artery diseasemedicineneurology

Citations

No citations found. To add a citation, contact the admin at info@scimatic.org

Comments

No comments yet. Be the first to comment on this article.