Neuroprotection of two C21 steroidal glycosides from against H2O2-induced damage on PC12 cells.

Clicks: 298
ID: 1425
2019
Article Quality & Performance Metrics
Overall Quality
Not rated
Combines reader engagement with the AI quality analysis. This article has not been analysed, so there is no overall score — reader engagement is measured and shown alongside.
AI Quality Assessment
Not analyzed
Readership in this journal
Steady

Ranked #29 of 83 articles by views in Natural product research

Most read Least read

Bar heights use a square-root scale.

Mint this article as an NFT
Not yet minted

Create a permanent, verifiable on-chain record of this article on the Scimatic Network. The NFT is held in your Journament account, and you can withdraw it to your own wallet at any time.

5 SUSD one-off · no wallet required
Sign in to mint
Abstract
Neuroprotection under conditions of oxygen stress of two C steroidal glycosides, cynsaccatol Q () and saccatol K (), isolated from Royle ex Wight, and their potential mechanisms were studied using the model of HO-induced damage on PC12 cells in this paper. Experiments showed that compounds and can both regulate the activities of antioxidant enzymes, including GSH-Px, LDH, CAT, SOD, decrease the levels of intracellular ROS and Ca, reduce cell apoptosis, and regulate the expression of HO-1 and NQO1 through the Nrf2-ARE pathway, thus play the role of neuroprotection against oxidative damage.
Reference Key
zhang2019neuroprotection Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Zhang, Mi;Wang, Dong;Li, Baocai;
Journal Natural product research
Year 2019
DOI
10.1080/14786419.2019.1636241
URL
Keywords Keywords not found

Citations

No citations found. To add a citation, contact the admin at info@scimatic.org

Comments

No comments yet. Be the first to comment on this article.