Peroxiredoxin II preserves cognitive function against age-linked hippocampal oxidative damage

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ID: 111842
2011
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Abstract
Reactive oxygen species (ROS), routinely produced in biological reactions, contribute to both normal aging and age-related decline in cognitive function. However, little is known regarding the involvement of specific antioxidants in the underlying mechanism(s). Here, we examined if peroxiredoxin II …
Reference Key
su2011neurobiologyperoxiredoxin Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Kim SU;Jin MH;Kim YS;Lee SH;Cho YS;Cho KJ;Lee KS;Kim YI;Kim GW;Kim JM;Lee TH;Lee YH;Shong M;Kim HC;Chang KT;Yu DY;Lee DS;;
Journal neurobiology of aging
Year 2011
DOI
DOI not found
URL
https://www.ncbi.nlm.nih.gov/pubmed/19577336
Keywords
Administration National Center for Biotechnology Information NCBI NLM MEDLINE Mice Inbred C57BL animals pubmed abstract nih national institutes of health national library of medicine behavior research support non-u.s. gov't male Animal Knockout microscopy electron pmid:19577336 doi:10.1016/j.neurobiolaging.2009.05.017 sun-uk kim mei-hua jin dong-seok lee oral aging / physiology* antioxidants / administration & dosage creb-binding protein / genetics creb-binding protein / metabolism cognition / drug effects cognition / physiology* cyclic amp-dependent protein kinases / metabolism enzyme activation / drug effects enzyme activation / genetics extracellular signal-regulated map kinases / metabolism hippocampus / cytology* learning disabilities / diet therapy learning disabilities / genetics long-term potentiation / drug effects long-term potentiation / genetics malondialdehyde / metabolism maze learning / physiology transmission / methods mitochondria / pathology neurons / drug effects neurons / metabolism neurons / physiology* neurons / ultrastructure peroxiredoxins / deficiency peroxiredoxins / metabolism* protein carbonylation / drug effects reactive oxygen species vitamin e / administration & dosage

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