Resveratrol protects cardiomyocytes against anoxia/reoxygenation via dephosphorylation of VDAC1 by Akt-GSK3 β pathway

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ID: 110499
2019
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Abstract
Our previous studies showed that the effect of resveratrol preventing mitochondrial permeability transition pore (mPTP) opening in myocardial ischemia/reperfusion injury was achieved by regulating voltage-dependent anion channel 1 (VDAC1). However, the underlying mechanism remains unclear. Previous …
Reference Key
m2019europeanresveratrol Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Tian M;Xie Y;Meng Y;Ma W;Tong Z;Yang X;Lai S;Zhou Y;He M;Liao Z;;
Journal European journal of pharmacology
Year 2019
DOI
DOI not found
URL
https://www.ncbi.nlm.nih.gov/pubmed/30445019##mesh-terms
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE animals pubmed abstract nih national institutes of health national library of medicine newborn pmid:30445019 doi:10.1016/j.ejphar.2018.11.016 Mengyuan Tian Yongyan Xie Zhangping Liao Apoptosis / drug effects Cardiotonic Agents / pharmacology* Cell Hypoxia / drug effects Cells Cultured Glycogen Synthase Kinase 3 beta / metabolism Myocytes Cardiac / drug effects* Cardiac / physiology Phosphorylation / drug effects Proto-Oncogene Proteins c-akt / metabolism Rats Sprague-Dawley Resveratrol / pharmacology* Voltage-Dependent Anion Channel 1 / metabolism

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