Resveratrol protects cardiomyocytes against anoxia/reoxygenation via dephosphorylation of VDAC1 by Akt-GSK3 β pathway

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ID: 110499
2019
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Abstract
Our previous studies showed that the effect of resveratrol preventing mitochondrial permeability transition pore (mPTP) opening in myocardial ischemia/reperfusion injury was achieved by regulating voltage-dependent anion channel 1 (VDAC1). However, the underlying mechanism remains unclear. Previous …
Reference Key
m2019europeanresveratrol Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Tian M, Xie Y, Meng Y, Ma W, Tong Z, Yang X, Lai S, Zhou Y, He M, Liao Z
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Journal European journal of pharmacology
Year 2019
DOI
DOI not found
URL
https://www.ncbi.nlm.nih.gov/pubmed/30445019##mesh-terms
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE animals pubmed abstract nih national institutes of health national library of medicine newborn pmid:30445019 doi:10.1016/j.ejphar.2018.11.016 Mengyuan Tian Yongyan Xie Zhangping Liao Apoptosis / drug effects Cardiotonic Agents / pharmacology* Cell Hypoxia / drug effects Cells Cultured Glycogen Synthase Kinase 3 beta / metabolism Myocytes Cardiac / drug effects* Cardiac / physiology Phosphorylation / drug effects Proto-Oncogene Proteins c-akt / metabolism Rats Sprague-Dawley Resveratrol / pharmacology* Voltage-Dependent Anion Channel 1 / metabolism

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