[1] Quantitative shedding patterns of respiratory syncytial virus in infants

[2] Analysis of the local and systemic immune responses induced in BALB/c mice by experimental respiratory syncytial virus infection

[3] Cytoplasmic inclusions of respiratory syncytial virus-infected cells: formation of inclusion bodies in transfected cells that coexpress the nucleoprotein, the phosphoprotein, and the 22K protein

[4] Transcription elongation factor of respiratory syncytial virus, a nonsegmented negative-strand RNA virus

[5] Role of T lymphocyte subsets in the pathogenesis of primary infection and rechallenge with respiratory syncytial virus in mice

[6] Antiviral properties of extract of Opuntia streptacantha

[7] Cytotoxic T cell activity against the 22-kDa protein of human respiratory syncytial virus (RSV) is associated with a significant reduction in pulmonary RSV replication

[8] Recombinant respiratory syncytial virus from which the entire SH gene has been deleted grows efficiently in cell culture and exhibits site-specific attenuation in the respiratory tract of the mouse

[9] Respiratory virus infection of peripheral blood monocytes: correlation with ageing of cells and interferon production in vitro

[10] The cell to cell infection of respiratory syncytial virus in HEp-2 monolayer cultures

[11] Immunoprophylaxis and immunotherapy of respiratory syncytial virus-infected mice with respiratory syncytial virus-specific immune serum

[12] Enhancement of respiratory syncytial virus pulmonary pathology in cotton rats by prior intramuscular inoculation of formalin-inactiva ted virus

[13] Immunoprophylaxis of respiratory syncytial virus infection in the infant ferret

[14] Pathogenesis and transmission of swine-origin 2009 A(H1N1) influenza virus in ferrets

[15] Delivery of antisense peptide nucleic acid by gold nanoparticles for the inhibition of virus replication.

[16] Activation of the autophagy pathway by Torovirus infection is irrelevant for virus replication.

[17] Combination therapy targeting platelet activation and virus replication protects mice against lethal influenza pneumonia.

[18] p-STAT1 regulates the influenza A virus replication and inflammatory response in vitro and vivo.

[19] Resveratrol affects Zika virus replication in vitro.

[20] Posaconazole inhibits dengue virus replication by targeting oxysterol-binding protein.

[21] Human Heat shock protein 40 (Hsp40/DnaJB1) promotes influenza A virus replication by assisting nuclear import of viral ribonucleoproteins

[22] Cinanserin is an inhibitor of the 3C-like proteinase of severe acute respiratory syndrome coronavirus and strongly reduces virus replication in vitro.

[23] Lithium chloride confers protection against viral myocarditis via suppression of coxsackievirus B3 virus replication.

[24] Human T Lymphocytes Are Permissive for Dengue Virus Replication

[25] Inhibitory effect of pyridobenzazoles on virus replication in vitro

[26] Human T Lymphocytes Are Permissive for Dengue Virus Replication

[27] Silibinin and SARS-CoV-2: Dual Targeting of Host Cytokine Storm and Virus Replication Machinery for Clinical Management of COVID-19 Patients

[28] Inhibition of respiratory syncytial virus replication by antisense oligodeoxyribonucleotides

[29] inhibition of hepatitis c virus replication and viral helicase by ethyl acetate extract of the marine feather star alloeocomatella polycladia

[30] cellular micrornas repress vesicular stomatitis virus but not theiler’s virus replication

[31] cytoskeletal dynamics: concepts in measles virus replication and immunomodulation

[32] initial influenza virus replication can be limited in allergic asthma through rapid induction of type iii interferons in respiratory epithelium

[33] chondrocytes contribute to alphaviral disease pathogenesis as a source of virus replication and soluble factor production

[34] pkr activation favors infectious pancreatic necrosis virus replication in infected cells

[35] inspirations on virus replication and cell-to-cell movement from studies examining the cytopathology induced by lettuce infectious yellows virus in plant cells

[36] Cutting edge: stealth influenza virus replication precedes the initiation of adaptive immunity

[37] Cutting Edge: Stealth Influenza Virus Replication Precedes the Initiation of Adaptive Immunity

[38] Quantitative aspects of inhibition of virus replication by interferon in chick embryo cell cultures

[39] IFN-I response timing relative to virus replication determines MERS coronavirus infection outcomes