[1] Limited Genetic Susceptibility to Severe Graves' Ophthalmopathy: No Role for CTLA-4 But Evidence for an Environmental Etiology

[2] Development and psychometric properties of the Thai Graves’ ophthalmopathy quality of life (GO-QOL) questionnaire

[3] research advances of orbital fibroblasts and thyroid associated ophthalmopathy

[4] graves ophthalmopathy: low-dose glucocorticoid increases peroxisome proliferator-activated receptor-gamma gene expression

[5] adiponectin concentration in the orbital fat of patients with graves' ophthalmopathy

[6] the influence of prior hyperthyroidism on euthyroid graves’ ophthalmopathy

[7] thickness of extraocular muscle and orbital fat in mri predicts response to glucocorticoid therapy in graves’ ophthalmopathy

[8] ct features of exophthalmos in chinese subjects with thyroid-associated ophthalmopathy

[9] inhibition effect of b7-h1 gene-modified regulatory dendritic cells on thyroid-associated ophthalmopathy in mice

[10] Limited Genetic Susceptibility to Severe Graves' Ophthalmopathy: No Role for CTLA-4 But Evidence for an Environmental Etiology