Peripheral vestibular disorders: an update.

Clicks: 419
ID: 58743
2019
Article Quality & Performance Metrics
Overall Quality
Not rated
Combines reader engagement with the AI quality analysis. This article has not been analysed, so there is no overall score — reader engagement is measured and shown alongside.
AI Quality Assessment
Not analyzed
Readership in this journal
Steady

Ranked #2 of 5 articles by views in current opinion in neurology

Most read Least read

Bar heights use a square-root scale.

Mint this article as an NFT
Not yet minted

Create a permanent, verifiable on-chain record of this article on the Scimatic Network. The NFT is held in your Journament account, and you can withdraw it to your own wallet at any time.

5 SUSD one-off · no wallet required
Sign in to mint
Abstract
To provide an update on the most frequent peripheral vestibular disorders.The on-going classification of vestibular disorders by the Bárány Society represents major progress. The diagnosis of bilateral vestibulopathy (BVP) requires quantitative testing of vestibular function. 'Acute unilateral peripheral vestibulopathy' (AUPVP) is now preferred over 'vestibular neuritis.' Menière's disease is a set of disorders with a significant genetic contribution. The apogeotropic variant of horizontal canal benign paroxysmal positional vertigo (hcBPPV) and anterior canal BPPV (acBPPV) can be distinguished from a central vestibular lesion. Vestibular paroxysmia is now an internationally accepted clinical entity. The diagnosis of SCDS is based on conclusive findings.Diagnosis of BVP requires significantly reduced vestibular function. The clinical picture of AUPVP depends on how much the vestibular end organs or their innervation are affected. Menière's disease phenotype is a constellation of symptoms. Although diagnostic and therapeutic criteria for pc and hcBPPV are well defined, a number of less frequent and controversial are increasingly diagnosed and can be treated. Diagnosis of vestibular paroxysmia requires that a patient responds to treatment with a sodium channel blocker. The diagnosis of SCDS requires conclusive findings with various methods. There is still a great need for state-of-the-art randomized controlled treatment trials in most peripheral vestibular disorders.
Reference Key
strupp2019peripheralcurrent Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Strupp, Michael;Mandalà, Marco;López-Escámez, Jose A;
Journal current opinion in neurology
Year 2019
DOI
10.1097/WCO.0000000000000649
URL
Keywords
glioma meningioma smarcb1 vestibular schwannoma acoustic neuroma cellular schwannoma central neurofibromatosis erm (ezrin/radixin/moesin) family scaffolding ependymoma epidemiology of familial tumor syndromes gardner glial hamartia glial micro-hamartoma intraneural schwannoma lztr1 manchester (nih) criteria manchester criteria meningioangiomatosis merlin nf2 neurofibroma neurofibromatosis type 2 neurofibromatosis type ii neurofibromin 2 plexiform schwannoma posterior subcapsular lenticular opacities sh3pxd2a-htra1 smarce1 sufu schwannoma schwannomatosis schwannomin verocay body von recklinghausen wishart

Citations

No citations found. To add a citation, contact the admin at info@scimatic.org

Comments

No comments yet. Be the first to comment on this article.