An essential role for the Zn transporter ZIP7 in B cell development.

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2019
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Abstract
Despite the known importance of zinc for human immunity, molecular insights into its roles have remained limited. Here we report a novel autosomal recessive disease characterized by absent B cells, agammaglobulinemia and early onset infections in five unrelated families. The immunodeficiency results from hypomorphic mutations of SLC39A7, which encodes the endoplasmic reticulum-to-cytoplasm zinc transporter ZIP7. Using CRISPR-Cas9 mutagenesis we have precisely modeled ZIP7 deficiency in mice. Homozygosity for a null allele caused embryonic death, but hypomorphic alleles reproduced the block in B cell development seen in patients. B cells from mutant mice exhibited a diminished concentration of cytoplasmic free zinc, increased phosphatase activity and decreased phosphorylation of signaling molecules downstream of the pre-B cell and B cell receptors. Our findings highlight a specific role for cytosolic Zn in modulating B cell receptor signal strength and positive selection.
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Authors Anzilotti, Consuelo;Swan, David J;Boisson, Bertrand;Deobagkar-Lele, Mukta;Oliveira, Catarina;Chabosseau, Pauline;Engelhardt, Karin R;Xu, Xijin;Chen, Rui;Alvarez, Luis;Berlinguer-Palmini, Rolando;Bull, Katherine R;Cawthorne, Eleanor;Cribbs, Adam P;Crockford, Tanya L;Dang, Tarana Singh;Fearn, Amy;Fenech, Emma J;de Jong, Sarah J;Lagerholm, B Christoffer;Ma, Cindy S;Sims, David;van den Berg, Bert;Xu, Yaobo;Cant, Andrew J;Kleiner, Gary;Leahy, T Ronan;de la Morena, M Teresa;Puck, Jennifer M;Shapiro, Ralph S;van der Burg, Mirjam;Chapman, J Ross;Christianson, John C;Davies, Benjamin;McGrath, John A;Przyborski, Stefan;Santibanez Koref, Mauro;Tangye, Stuart G;Werner, Andreas;Rutter, Guy A;Padilla-Parra, Sergi;Casanova, Jean-Laurent;Cornall, Richard J;Conley, Mary Ellen;Hambleton, Sophie;
Journal Nature Immunology
Year 2019
DOI
10.1038/s41590-018-0295-8
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