Can long-term bisoprolol therapy trigger hypothyroidism?
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ID: 311515
2024
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Abstract
Bisoprolol is a selective beta1-blocker medication utilized for the management of hypertension, congestive heart failure, and chronic stable angina. Its administration is associated with a reduction in morbidity and mortality following myocardial infarction, as well as a decreased risk of stroke and coronary artery disease in individuals with heart conditions. 1,2 Bisoprolol, along with other selective Beta1-blockers, can lead to negative impacts on the heart’s contractions and rate. As a result, bisoprolol diminishes the oxygen demand of myocardial cells, alleviating the workload on the heart and subsequently contributing to a reduction in morbidity and mortality rates after a myocardial infarction. 2 Considering the data indicating that prolonged use of beta blockers influences serum levels of T4, T3, and rT3 in euthyroid patients suffering from ischemic heart disease, 3 it is pertinent to inquire whether long-term beta blocker therapy may trigger hypothyroidism in these individuals. In this context, we present a case involving a male patient who was diagnosed with ischemic heart disease and later developed hypothyroidism following prolonged treatment with bisoprolol.
| Reference Key |
mussian2024longterm
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|---|---|
| Authors | Nabeel Salim Mussian, Ahmed Salem Basabbea |
| Journal | Yemen Journal of Medicine |
| Year | 2024 |
| DOI |
10.18231/j.yjom.2024.035
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| URL | |
| Keywords | Keywords not found |
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