Universal definition of myocardial infarction: Kristian Thygesen, Joseph S. Alpert and Harvey D. White on behalf of the Joint ESC/ACCF/AHA/WHF Task Force for the Redefinition of Myocardial Infarction
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ID: 290404
2007
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Abstract
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Myocardial infarction is a major cause of death and disability worldwide. Coronary atherosclerosis is a chronic disease with stable and unstable periods. During unstable periods with activated inflammation in the vascular wall, patients may develop a myocardial infarction. Myocardial infarction may be a minor event in a lifelong chronic disease, it may even go undetected, but it may also be a major catastrophic event leading to sudden death or severe haemodynamic deterioration. A myocardial infarction may be the first manifestation of coronary artery disease, or it may occur, repeatedly, in patients with established disease. Information on myocardial infarction attack rates can provide useful data regarding the burden of coronary artery disease within and across populations, especially if standardized data are collected in a manner that demonstrates the distinction between incident and recurrent events. From the epidemiological point of view, the incidence of myocardial infarction in a population can be used as a proxy for the prevalence of coronary artery disease in that population. Furthermore, the term myocardial infarction has major psychological and legal implications for the individual and society. It is an indicator of one of the leading health problems in the world, and it is an outcome measure in clinical trials and observational studies. With these perspectives, myocardial infarction may be defined from a number of different clinical, electrocardiographic, biochemical, imaging, and pathological characteristics.
In the past, a general consensus existed for the clinical syndrome designated as myocardial infarction. In studies of disease prevalence, the World Health Organization (WHO) defined myocardial infarction from symptoms, ECG abnormalities, and enzymes. However, the development of more sensitive and specific serological biomarkers and precise imaging techniques allows detection of ever smaller amounts of myocardial necrosis. Accordingly, current clinical practice, health care delivery systems, as well as epidemiology and clinical trials all require a …
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| Authors | K. Thygesen, Joseph S. Alpert, Harvey D. White, Allan S. Jaffe, Fred S. Apple, Marcello Galvani, Hugo A. Katus, L. Kristin Newby, Jan Ravkilde, Bernard Chaitman, Peter Clemmensen, Mikael Dellborg, Hanoch Hod, Pekka Porela, R. Underwood, Jeroen J. Bax, G A Beller, Robert O. Bonow, Ernst E. van der Wall, Jean‐Pierre Bassand, William Wijns, T. Bruce Ferguson, Philippe Gabríel Steg, Barry F. Uretsky, Devan Williams, Paul W. Armstrong, Elliott M. Antman, Keith A.A. Fox, C Hamm, E. Magnus Ohman, Maarten L. Simoons, P A Poole-Wilson, Enrique Gurfinkel, J L López-Sendón, Priya Pais, Shanthi Mendis, Jiaying Zhu, Lars Wallentin, Francisco Fernández‐Avilés, K M Fox, А. Н. Пархоменко, S. G. Priori, Michał Tendera, L M Voipio-Pulkki, A. Vahanian, A. J. Camm, Raffaele De Caterina, V. Dean, Kenneth Dickstein, G. Filippatos, Christian Funck‐Brentano, Irene Hellemans, Salome Kristensen, K. McGregor, Udo Sechtem, Sigmund Silber, Michał Tendera, P. Widimsky, José Luis Zamorano, João Morais, S. Brener, Robert A. Harrington, David A. Morrow, Udo Sechtem, Mervyn Jun Rui Lim, Marco Antonio Martínez‐Ríos, Steven R. Steinhubl, Gregory N. Levine, W. Brian Gibler, David C. Goff, M. Tubaro, D Dudek, Nawwar Al‐Attar |
| Journal | european heart journal |
| Year | 2007 |
| DOI |
10.1093/eurheartj/ehm355
|
| URL | |
| Keywords | Keywords not found |
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