Early enhanced Th1 response after Leishmania amazonensis infection of C57BL/6 interleukin-10-deficient mice does not lead to resolution of infection

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ID: 274226
2002
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Abstract
C3H and C57BL/6 mice are resistant to Leishmania major but develop chronic lesions with persistent parasite loads when they are infected with Leishmania amazonensis. These lesions develop in the absence of interleukin-4 (IL-4), indicating that susceptibility to this parasite is not a result of devel …
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de2002infectionearly Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Jones DE;Ackermann MR;Wille U;Hunter CA;Scott P;;
Journal Infection and immunity
Year 2002
DOI
DOI not found
URL
http://www.ncbi.nlm.nih.gov/pubmed/11895981
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE Mice Inbred C57BL animals pubmed abstract nih national institutes of health national library of medicine research support non-u.s. gov't u.s. gov't P.H.S. Antibodies hypersensitivity immunoglobulin g / classification interferon-gamma / biosynthesis delayed protozoan / biosynthesis leishmaniasis cutaneous / immunology* th1 cells / immunology* nitric oxide synthase / genetics nitric oxide synthase type ii interleukin-10 / physiology* interleukin-12 / biosynthesis pmid:11895981 pmc127855 doi:10.1128/iai.70.4.2151-2158.2002 douglas e jones mark r ackermann phillip scott leishmania major leishmania mexicana / immunology* cutaneous / pathology

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