miR-101a and miR-30b contribute to inflammatory cytokine-mediated β-cell dysfunction

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ID: 273845
2015
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Ranked #6 of 7 articles by views in laboratory investigation; a journal of technical methods and pathology

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Abstract
Inflammatory cytokines have a critical role in the progressive deterioration of pancreatic β-cell function and development of type 1 diabetes. Prolonged exposure of β-cells to inflammatory cytokines results in gene expression modifications, leading to loss of β-cell function. MicroRNAs (miRNAs) are …
Reference Key
y2015laboratorymir-101a Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Zheng Y;Wang Z;Tu Y;Shen H;Dai Z;Lin J;Zhou Z;;
Journal laboratory investigation; a journal of technical methods and pathology
Year 2015
DOI
DOI not found
URL
https://www.ncbi.nlm.nih.gov/pubmed/26367486
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE animals humans pubmed abstract nih national institutes of health national library of medicine research support non-u.s. gov't cytokines micrornas / metabolism* transcription factors / metabolism apoptosis diabetes mellitus hek293 cells insulin / biosynthesis insulin-secreting cells / metabolism* ying zheng zhen wang zhiguang zhou homeodomain proteins / metabolism basic helix-loop-helix transcription factors / metabolism pmid:26367486 doi:10.1038/labinvest.2015.112 type 1 / etiology* proto-oncogene proteins c-bcl-2 / metabolism

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