PPM1D dephosphorylates Chk1 and p53 and abrogates cell cycle checkpoints
Clicks: 208
ID: 272881
2005
Article Quality & Performance Metrics
Overall Quality
Not rated
Combines reader engagement with the AI quality analysis. This
article has not been analysed, so there is no overall score —
reader engagement is measured and shown alongside.
Reader Engagement
Steady Performance
30.0
/100
208 views
18 readers
AI Quality Assessment
Not analyzed
Readership in this journal
SteadyRanked #36 of 206 articles by views in genes & development
Most read
Least read
Bar heights use a square-root scale. Only the 120 most-read articles are drawn; the journal has 206 in total.
Mint this article as an NFT
Not yet mintedCreate a permanent, verifiable on-chain record of this article on the Scimatic Network. The NFT is held in your Journament account, and you can withdraw it to your own wallet at any time.
5
SUSD
one-off · no wallet required
Abstract
The ATM (ataxia-telangiectasia mutated) and ATR (ataxia-telangiectasia and Rad3-related) kinases respond to DNA damage by phosphorylating cellular target proteins that activate DNA repair pathways and cell cycle checkpoints in order to maintain genomic ...
Abstract Quality Issue:
This abstract appears to be incomplete or contains metadata (34 words).
Try re-searching for a better abstract.
| Reference Key |
donehower2005genesppm1d
Use this key to autocite in the manuscript while using
SciMatic Manuscript Manager or Thesis Manager
|
|---|---|
| Authors | Xiongbin Lu, Bonnie Nannenga, Lawrence A. Donehower;Xiongbin Lu;Bonnie Nannenga;Lawrence A. Donehower; |
| Journal | genes & development |
| Year | 2005 |
| DOI |
10.1101/gad.1291305
|
| URL | |
| Keywords |
|
Citations
No citations found. To add a citation, contact the admin at info@scimatic.org
Comments
No comments yet. Be the first to comment on this article.