Systemic antibodies towards mucosal bacteria in ulcerative colitis and Crohn's disease differentially activate the innate immune response

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ID: 272552
2004
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Abstract
Changes in mucosal bacteria, and a switch from internal to surface antigen/antibody reactivity of a predominantly IgG1 type, leads to greater opsonisation of the respiratory burst in PMN, providing a mechanism for maintaining the inflammatory state in UC.
Reference Key
e2004gutsystemic Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Furrie E, Macfarlane S, Cummings JH, Macfarlane GT
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Journal Gut
Year 2004
DOI
DOI not found
URL
http://www.ncbi.nlm.nih.gov/pubmed/14684582
Keywords
immunity National Center for Biotechnology Information NCBI NLM MEDLINE humans pubmed abstract nih national institutes of health national library of medicine research support non-u.s. gov't female male aged middle aged 80 and over Antibodies Antigens Bacteria / isolation & purification* intestinal mucosa / immunology intestinal mucosa / microbiology* Colitis immunoglobulin g / blood bacterial / immunology* bacterial / blood bacterial / immunology surface / immunology neutrophils / immunology g t macfarlane ulcerative / microbiology immunoglobulin a / blood bacteria / immunology rectum / microbiology crohn disease / microbiology ulcerative / immunology* pmid:14684582 pmc1773925 doi:10.1136/gut.53.1.91 e furrie s macfarlane crohn disease / immunology* mucosal rectum / immunology respiratory burst

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