Sulforaphane enhances the activity of the Nrf2–ARE pathway and attenuates inflammation in OxyHb-induced rat vascular smooth muscle cells

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ID: 271118
2013
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Abstract
A growing body of evidence indicates that the nuclear factor erythroid 2-related factor 2–antioxidant response element (Nrf2–ARE) pathway plays a protective role in many physiological stress processes such as inflammatory damage, oxidative stress, and the accumulation of toxic metabolites, which are all involved in the cerebral vasospasm following subarachnoid hemorrhage (SAH). We hypothesized that the Nrf2–ARE pathway might have a protective role in cerebral vasospasm following SAH.
Reference Key
lu2013inflammationsulforaphane Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors X.-D. Zhao,Y.-T. Zhou,X.-J. Lu;X.-D. Zhao;Y.-T. Zhou;X.-J. Lu;
Journal inflammation research
Year 2013
DOI
10.1007/s00011-013-0641-0
URL
https://sciprofiles.com/publication/view/29ebf6f616f389f7c5427262cb50be61
Keywords
subarachnoid hemorrhage nrf2 heme oxygenase-1 inflammatory cerebral vasospasm vsmcs oxyhb

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