Genetic rescue of Muenke syndrome model hearing loss reveals prolonged FGF-dependent plasticity in cochlear supporting cell fates
Clicks: 179
ID: 270639
2013
Article Quality & Performance Metrics
Overall Quality
Not rated
Combines reader engagement with the AI quality analysis. This
article has not been analysed, so there is no overall score —
reader engagement is measured and shown alongside.
Reader Engagement
Emerging Content
30.0
/100
179 views
23 readers
AI Quality Assessment
Not analyzed
Readership in this journal
EmergingRanked #55 of 206 articles by views in genes & development
Most read
Least read
Bar heights use a square-root scale. Only the 120 most-read articles are drawn; the journal has 206 in total.
Mint this article as an NFT
Not yet mintedCreate a permanent, verifiable on-chain record of this article on the Scimatic Network. The NFT is held in your Journament account, and you can withdraw it to your own wallet at any time.
5
SUSD
one-off · no wallet required
Abstract
The stereotyped arrangement of cochlear sensory and supporting cells is critical for auditory function. Our previous studies showed that Muenke syndrome model mice (Fgfr3[P244R/+] ) have hearing loss associated with a supporting cell fate transformation ...
Abstract Quality Issue:
This abstract appears to be incomplete or contains metadata (36 words).
Try re-searching for a better abstract.
| Reference Key |
urness2013genesgenetic
Use this key to autocite in the manuscript while using
SciMatic Manuscript Manager or Thesis Manager
|
|---|---|
| Authors | Suzanne L. Mansour, Chaoying Li, Lisa D. Urness;Suzanne L. Mansour;Chaoying Li;Lisa D. Urness; |
| Journal | genes & development |
| Year | 2013 |
| DOI |
10.1101/gad.228957.113
|
| URL | |
| Keywords |
|
Citations
No citations found. To add a citation, contact the admin at info@scimatic.org
Comments
No comments yet. Be the first to comment on this article.