Fetal programming: excess prenatal testosterone reduces postnatal luteinizing hormone, but not follicle-stimulating hormone responsiveness, to estradiol negative feedback in the female

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ID: 269950
2005
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Abstract
Exposure of female sheep fetuses to excess testosterone (T) during early to midgestation produces postnatal hypergonadotropism manifest as a selective increase in LH. This hypergonadotropism may result from reduced sensitivity to estradiol (E2) negative feedback and/or increased pituitary sensitivit …
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hn2005endocrinologyfetal Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Sarma HN, Manikkam M, Herkimer C, Dell'Orco J, Welch KB, Foster DL, Padmanabhan V
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Journal endocrinology
Year 2005
DOI
DOI not found
URL
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Abstract&list_uids=15976056
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE animals pubmed abstract nih national institutes of health national library of medicine pregnancy research support non-u.s. gov't u.s. gov't female male N.I.H. Extramural P.H.S. luteinizing hormone / metabolism* sexual maturation / drug effects testosterone / pharmacology* feedback sheep follicle stimulating hormone / pharmacology* fetus / drug effects estradiol / administration & dosage estradiol / pharmacology* pmid:15976056 doi:10.1210/en.2005-0322 hirendra n sarma mohan manikkam vasantha padmanabhan drug implants fetus / physiology*

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