PTEN mutations do not cause nuclear beta-catenin accumulation in endometrial carcinomas

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ID: 266810
2004
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Abstract
PTEN: and beta-catenin mutations constitute the predominant genetic alterations in endometrioid carcinomas of the endometrium. PTEN encodes a dual-specificity phosphatase with lipid phosphatase and protein tyrosine phosphatase activities that regulate both apoptosis and interactions with the extrace …
Reference Key
b2004humanpten Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Wappenschmidt B;Wardelmann E;Gehrig A;Schöndorf T;Maass N;Bonatz G;Gassel AM;Pietsch T;Mallmann P;Weber BH;Schmutzler RK;;
Journal human pathology
Year 2004
DOI
DOI not found
URL
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Abstract&list_uids=15492994
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE humans pubmed abstract nih national institutes of health national library of medicine research support non-u.s. gov't female carcinoma immunohistochemistry mutation* trans-activators / metabolism* cell nucleus / metabolism pten phosphohydrolase beta catenin pmid:15492994 doi:10.1016/j.humpath.2004.06.007 b wappenschmidt e wardelmann r k schmutzler endometrioid / genetics* endometrioid / metabolism* endometrioid / pathology cytoskeletal proteins / metabolism* dna mutational analysis endometrial neoplasms / genetics* endometrial neoplasms / metabolism* endometrial neoplasms / pathology phosphoric monoester hydrolases / genetics* tumor suppressor proteins / genetics*

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