PTEN mutations do not cause nuclear beta-catenin accumulation in endometrial carcinomas

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ID: 266810
2004
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Abstract
PTEN: and beta-catenin mutations constitute the predominant genetic alterations in endometrioid carcinomas of the endometrium. PTEN encodes a dual-specificity phosphatase with lipid phosphatase and protein tyrosine phosphatase activities that regulate both apoptosis and interactions with the extrace …
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b2004humanpten Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Wappenschmidt B, Wardelmann E, Gehrig A, Schöndorf T, Maass N, Bonatz G, Gassel AM, Pietsch T, Mallmann P, Weber BH, Schmutzler RK
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Journal human pathology
Year 2004
DOI
DOI not found
URL
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Abstract&list_uids=15492994
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE humans pubmed abstract nih national institutes of health national library of medicine research support non-u.s. gov't female carcinoma immunohistochemistry mutation* trans-activators / metabolism* cell nucleus / metabolism pten phosphohydrolase beta catenin pmid:15492994 doi:10.1016/j.humpath.2004.06.007 b wappenschmidt e wardelmann r k schmutzler endometrioid / genetics* endometrioid / metabolism* endometrioid / pathology cytoskeletal proteins / metabolism* dna mutational analysis endometrial neoplasms / genetics* endometrial neoplasms / metabolism* endometrial neoplasms / pathology phosphoric monoester hydrolases / genetics* tumor suppressor proteins / genetics*

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