alternative neural circuitry that might be impaired in the development of alzheimer disease

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ID: 255934
2015
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Abstract
It is well established that some individuals with normal cognitive capacity have abundant senile plaques in their brains. It has been proposed that those individuals are resilient or have compensation factors to prevent cognitive decline. In this comment, we explore an alternative mechanism through which cognitive capacity is maintained. This mechanism could involve the impairment of alternative neural circuitry. Also, the proportion of molecules such as Aβ or tau protein present in different areas of the brain could be important.
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eavila2015frontiersalternative Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors ;Jesus eAvila;Jesus eAvila;George ePerry;Bryan Andrew Strange;Bryan Andrew Strange;Felix eHernandez;Felix eHernandez
Journal Journal of enzyme inhibition and medicinal chemistry
Year 2015
DOI
10.3389/fnins.2015.00145
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