progress in research of nogo after optic nerve injury

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ID: 215383
2013
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Abstract
After mammalian peripheral nerve injury, axons can regenerate very long distance. In certain conditions, the central nervous system can also partially regenerate after damage. Nogo has been proved to be one of the myelin-associated inhibitors, after central nerve injury increased Nogo release, enhanced Nogo expression, start-up process of apoptosis, leading to neuronal death. In this paper, we summed up the mechanism of action of Nogo from Nogo-A, Nogo-66, soluble NgR fragments, RhoA enzyme and Rho-A/Rho kinase signaling pathway, calcium ion aspects, and discussed its application prospect in nerve regeneration after optic nerve injury.
Reference Key
tang2013guojiprogress Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors ;Yong-Ying Tang;Lin-Kun Ma;Xia Cao
Journal journal of fungi
Year 2013
DOI
10.3980/j.issn.1672-5123.2013.09.12
URL
http://www.ies.net.cn/cn_publish/2013/9/201309012.pdf
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