Internalization of Constitutively Active N111G Mutant of AT1 Receptor Induced by Angiotensin II–Receptor Antagonists Candesartan, Losartan, and Telmisartan: Comparison With Valsartan

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ID: 18829
2010
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Abstract
Based on radioligand binding and signal transduction assays in our previous study, we have determined the binding pattern and functional efficacy of the constitutively active mutant N111G of angiotensin II type 1 (AT1) receptor. We have also shown that the N111G mutant induces homologous internalization through mediation of the AT1-receptor antagonist valsartan. In this study we demonstrated that other AT1-receptor antagonists, candesartan, losartan, and telmisartan, also stimulate internalization of N111G mutant receptor to the same extent. We further showed that the internalization pattern is also similar for all the AT1-receptor antagonists. Keywords:: angiotensin II type 1 (AT1) receptor, constitutively active mutant, internalization
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bhuiyan2010internalizationjournal Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Bhuiyan, Mohiuddin Ahmed;Hossain, Murad;Nakamura, Takashi;Ozaki, Masanobu;Nagatomo, Takafumi;
Journal Journal of pharmacological sciences
Year 2010
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