therapeutically targeting neuroinflammation and microglia after acute ischemic stroke

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ID: 148564
2014
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Abstract
Inflammation has a pivotal role in the pathogenesis of ischemic stroke, and recent studies posit that inflammation acts as a double-edged sword, not only detrimentally augmenting secondary injury, but also potentially promoting recovery. An initial event of inflammation in ischemic stroke is the activation of microglia, leading to production of both pro- and anti-inflammatory mediators acting through multiple receptor signaling pathways. In this review, we discuss the role of microglial mediators in acute ischemic stroke and elaborate on preclinical and clinical studies focused on microglia in stroke models. Understanding how microglia can lead to both pro- and anti-inflammatory responses may be essential to implement therapeutic strategies using immunomodulatory interventions in ischemic stroke.
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lee2014biomedtherapeutically Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors ;Youngjeon Lee;Sang-Rae Lee;Sung S. Choi;Hyeon-Gu Yeo;Kyu-Tae Chang;Hong J. Lee
Journal spectrochimica acta - part a: molecular and biomolecular spectroscopy
Year 2014
DOI
10.1155/2014/297241
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