aβ-induced vulnerability propagates via the brain’s default mode network
Clicks: 235
ID: 131351
2019
Article Quality & Performance Metrics
Overall Quality
Not rated
Combines reader engagement with the AI quality analysis. This
article has not been analysed, so there is no overall score —
reader engagement is measured and shown alongside.
Reader Engagement
Steady Performance
30.0
/100
235 views
24 readers
AI Quality Assessment
Not analyzed
Readership in this journal
SteadyRanked #8 of 39 articles by views in educacao e sociedade
Most read
Least read
Bar heights use a square-root scale.
Mint this article as an NFT
Not yet mintedCreate a permanent, verifiable on-chain record of this article on the Scimatic Network. The NFT is held in your Journament account, and you can withdraw it to your own wallet at any time.
5
SUSD
one-off · no wallet required
Abstract
Amyloid-β (Aβ) deposition occurs in Alzheimer's disease but its relation to disease features such as local brain hypometabolism or cognitive decline is unclear. Here, the authors show that Aβ aggregation in the brain’s default mode network leads to hypometabolism in distant but functionally connected areas.
| Reference Key |
pascoal2019naturea-induced
Use this key to autocite in the manuscript while using
SciMatic Manuscript Manager or Thesis Manager
|
|---|---|
| Authors | ;Tharick A. Pascoal;Sulantha Mathotaarachchi;Min Su Kang;Sara Mohaddes;Monica Shin;Ah Yeon Park;Maxime J. Parent;Andrea L. Benedet;Mira Chamoun;Joseph Therriault;Heungsun Hwang;A. Claudio Cuello;Bratislav Misic;Jean-Paul Soucy;John A. D. Aston;Serge Gauthier;Pedro Rosa-Neto |
| Journal | educacao e sociedade |
| Year | 2019 |
| DOI |
10.1038/s41467-019-10217-w
|
| URL | |
| Keywords |
Citations
No citations found. To add a citation, contact the admin at info@scimatic.org
Comments
No comments yet. Be the first to comment on this article.