Microglia play a role in ethanol-induced oxidative stress and apoptosis in developing hypothalamic neurons

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ID: 120061
2013
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Abstract
These data suggest that exposure of developing hypothalamic neurons to EtOH increases cellular apoptosis via the effects on oxidative stress of neurons directly and via increasing production of microglial-derived factor(s).
Reference Key
ni2013alcoholism,microglia Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Boyadjieva NI;Sarkar DK;;
Journal Alcoholism, clinical and experimental research
Year 2013
DOI
DOI not found
URL
https://www.ncbi.nlm.nih.gov/pubmed/22823548
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE animals pubmed abstract nih national institutes of health national library of medicine pregnancy research support female N.I.H. Extramural reactive oxygen species / metabolism Rats Sprague-Dawley culture media dose-response relationship drug apoptosis / drug effects* neurons / drug effects antioxidants / pharmacology chromans / pharmacology ethanol / adverse effects* ethanol / antagonists & inhibitors fetus / drug effects* fetus / metabolism pmid:22823548 pmc3481017 doi:10.1111/j.1530-0277.2012.01889.x nadka i boyadjieva dipak k sarkar antioxidants / metabolism conditioned / pharmacology hypothalamus / metabolism* hypothalamus / physiopathology microglia / metabolism* neurons / physiology organometallic compounds / pharmacology oxidative stress / drug effects* primary cell culture salicylates / pharmacology

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