Mitochondrial energy metabolism and redox signaling in brain aging and neurodegeneration

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ID: 116186
2014
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Abstract
Characterization of mitochondrial function and mitochondria-cytosol communications will provide pivotal opportunities for identifying targets and developing new strategies aimed at restoring the mitochondrial energy-redox axis that is compromised in brain aging and neurodegeneration.
Reference Key
f2014antioxidantsmitochondrial Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Yin F;Boveris A;Cadenas E;;
Journal antioxidants & redox signaling
Year 2014
DOI
DOI not found
URL
https://www.ncbi.nlm.nih.gov/pubmed/22793257
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE genetic review gene expression regulation animals humans pubmed abstract nih national institutes of health national library of medicine research support N.I.H. Extramural Cell Nucleus / genetics transcription signal transduction neurodegenerative diseases / genetics homeostasis energy metabolism* oxidation-reduction brain / metabolism* pmid:22793257 pmc3887431 doi:10.1089/ars.2012.4774 fei yin alberto boveris enrique cadenas aging / metabolism* cell nucleus / metabolism cellular senescence cytosol / metabolism mitochondria / metabolism* neurodegenerative diseases / metabolism*

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