Does the Nitric Oxide Synthase T786C Gene Polymorphism Influence Arterial Stiffness in Patients with Metabolic Syndrome?

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ID: 113800
1970
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Abstract
Background: Endothelial Nitric Oxide Synthase (eNOS) is responsible for Nitric Oxide (NO) bioavailability at endothelial level. Aging (even in healthy people) is involved in arterial stiffness increases. Materials and Methods: We investigated (in the service of Cardiology, 4th Medical Clinic) 100 patients, 55 with metabolic syndrome (MS), mean age...
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cozma1970arterydoes Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Angela Cozma;Adriana Fodor;Lucia Maria Procopciuc;Teodora Alexescu;Vasile Negrean;Dana Pop;Doina Alina Todea;Olga Hilda Orăşan;Adela-Viviana Sitar-Taut;Angela Cozma;Adriana Fodor;Lucia Maria Procopciuc;Teodora Alexescu;Vasile Negrean;Dana Pop;Doina Alina Todea;Olga Hilda Orăşan;Adela-Viviana Sitar-Taut;
Journal artery research
Year 1970
DOI
DOI not found
URL
https://doi.org/10.2991/artres.k.200324.001
Keywords
enos gene polymorphisms t786c arterial stiffness metabolic syndrome

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