Developmental programming: impact of prenatal testosterone excess on insulin sensitivity, adiposity, and free fatty acid profile in postpubertal female sheep

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ID: 111463
2013
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Abstract
Prenatal T excess causes reproductive and metabolic disruptions including insulin resistance, attributes of women with polycystic ovary syndrome. This study tested whether increases in visceral adiposity, adipocyte size, and total free fatty acids underlie the insulin resistance seen in prenatal T-t …
Reference Key
a2013endocrinologydevelopmental Use this key to autocite in the manuscript while using SciMatic Manuscript Manager or Thesis Manager
Authors Veiga-Lopez A;Moeller J;Patel D;Ye W;Pease A;Kinns J;Padmanabhan V;;
Journal endocrinology
Year 2013
DOI
DOI not found
URL
http://www.ncbi.nlm.nih.gov/pubmed/23525243
Keywords
National Center for Biotechnology Information NCBI NLM MEDLINE animals pubmed abstract nih national institutes of health national library of medicine pregnancy research support female N.I.H. Extramural sex factors Fatty Acids pmid:23525243 pmc4016698 doi:10.1210/en.2012-2145 a veiga-lopez j moeller v padmanabhan adiposity / drug effects* embryonic development / drug effects* embryonic development / physiology* nonesterified / metabolism* insulin resistance* / physiology metabolome / drug effects prenatal exposure delayed effects* / chemically induced prenatal exposure delayed effects* / metabolism prenatal exposure delayed effects* / physiopathology sexual maturation / drug effects sexual maturation / physiology sheep / embryology sheep / growth & development sheep / metabolism sheep / physiology testosterone / pharmacology*

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