Effects of Glucagon-Like Peptide-1 on Oxidative Stress and Nrf2 Signaling
Clicks: 533
ID: 110075
2017
Article Quality & Performance Metrics
Overall Quality
Improving Quality
0.0
/100
Combines engagement data with AI-assessed academic quality
Reader Engagement
Popular Article
30.0
/100
531 views
59 readers
Trending
AI Quality Assessment
Not analyzed
Mint this article as an NFT
Not yet mintedCreate a permanent, verifiable on-chain record of this article on the Scimatic Network. The NFT is held in your Journament account, and you can withdraw it to your own wallet at any time.
5
SUSD
one-off · no wallet required
Abstract
Oxidative cellular damage caused by free radicals is known to contribute to the pathogenesis of various diseases such as cancer, diabetes, and neurodegenerative diseases, as well as to aging. The transcription factor nuclear factor erythroid 2-related factor 2 (Nrf2) and Kelch-like ECH-associated pr …
Abstract Quality Issue:
This abstract appears to be incomplete or contains metadata (42 words).
Try re-searching for a better abstract.
| Reference Key |
ys2017internationaleffects
Use this key to autocite in the manuscript while using
SciMatic Manuscript Manager or Thesis Manager
|
|---|---|
| Authors | Oh YS;Jun HS;; |
| Journal | International journal of molecular sciences |
| Year | 2017 |
| DOI |
DOI not found
|
| URL | |
| Keywords |
National Center for Biotechnology Information
NCBI
NLM
MEDLINE
review
animals
humans
pubmed abstract
nih
national institutes of health
national library of medicine
pmid:29271910
pmc5795977
doi:10.3390/ijms19010026
yoon sin oh
hee-sook jun
diabetes mellitus / metabolism
glucagon-like peptide 1 / metabolism*
nf-e2-related factor 2 / metabolism*
nervous system diseases / metabolism
oxidative stress*
reactive oxygen species / metabolism
signal transduction*
Diabetes
oxidative stress
reactive oxygen species
glucagon-like peptide-1
nrf2 signaling
|
Citations
No citations found. To add a citation, contact the admin at info@scimatic.org
Comments
No comments yet. Be the first to comment on this article.